Amoebic Dysentery

Amoebic dysentery-What is amoebic dysentery?

Amoebic dysentery (amebicdysentery), also known as intestinal amebiasis (intestinalamebiasis), by pathogenic Entamoeba histolytica protozoa invade the colon wall caused by gastrointestinal diseases mainly dysentery symptoms. Lesions in the colon, ileocecal easy to relapse becomes chronic. The protozoa also by the intestinal wall by the blood flow - lymphatic or direct migration to the liver, lung, brain and other organs become intestinal amoebiasis, especially amoebic liver abscess is most prevalent.
Edit SummaryAmoebic dysentery - Basic OverviewFor the body only pathogenic amoeba, amoebic dysentery dysentery amoeba (dissolved histolytica, Amoebahistolytica) in human tissues and faeces large trophozoites the small trophozoites and cysts of three forms. Trophozoites in vitro resistance to weak, easy death. Cysts strong resistance to the outside world.(A) the size of 20 ~ 40μm the trophozoites large trophozoites rely pseudopodia certain direction, found in the feces or intestinal tissue of patients with acute swallowed tissues and erythrocytes, it is also known as tissue-type trophozoites. Small trophozoites 6 ~ 20μm size, pseudopodia, to host intestinal fluid, bacteria, fungi as food, do not eat red blood cells, also known as the intestine-type trophozoites. When host declining health, the secretion of soluble tissue enzymes, combined with their own movement and invade the intestinal submucosa becomes large trophozoites; into cysts pre intestine conditions change a negative effect on their activities, and then turn into cysts. The trophozoites dissemination.
(B) cysts are more common in the feces of latent infection and chronic patients, rounded, 5 ~ 20μm size, mature cysts have four nuclear Entamoeba histolytica infection type, infectious. Cysts in the stool on the outside resistant survive for at least 2 weeks, 5 weeks of water, the refrigerator 2 months, more resistant to chemical disinfectants, can tolerate 0.2% potassium permanganate for several days, the ordinary drinking disinfecting chlorine concentration of its no role in the killing, but the heat (50 ℃) and dry and very sensitive.
Entamoeba histolytica culture bacteria was symbiosis. A symbiotic culture has been successful, provided the conditions for the the pure antigen preparation and in-depth research Entamoeba histolytica.
Amoebic dysentery - EpidemiologyAmoebic dysentery and chronic patients, convalescent patients and cyst carriers is the main source of infection of the disease. Gastrointestinal spread through contaminated water, vegetables, fruits and food can also be spread indirectly through contaminated hands, supplies flies, cockroaches and other oral. Ordinary population susceptible to infection immunity (ie, does not produce protective antibodies), it is easy to re-infection. Of the disease throughout the world, prevalent in tropical and subtropical regions. China is more common in the north. The incidence rate in rural areas than in urban; men than in women, adults more often than children, most of them are sporadic, even outbreaks due to water pollution and other factors.Pathogenesis and pathological changes: amoeba cysts into the digestive tract, the lower section of the small intestine is trypsin and other digestive juices to digest, parasite excystation escape, and repeatedly divide to form the majority of small trophozoites, a stranger in the land of the ileocecal colon parts of the health of the host medium and small trophozoites with feces down below to sigmoid becomes cysts excreted, not pathogenic. Under suitable conditions, such as gastrointestinal function of the body is reduced; certain bacterial episome-like factor, enhanced virulence nourishing; trophozoites release of lysosomal enzymes, hyaluronidase, proteolytic enzymes and rely on its pseudopodia mechanical activity, invade the intestinal mucosa, destroying tissue formation of small abscesses and latent form (beaker-like) ulcers, deep muscle, causing extensive tissue damage trophozoites with necrotic material and blood from the intestinal tract, showing symptoms of dysentery.
Chronic changes in the mucosal epithelial hyperplasia, ulcers formed at the bottom of the granulation tissue around the ulcer, see the fibrous tissue hyperplasia, the formation of intestinal amebiasis. Trophozoites may also enter the intestinal wall vein into the liver through the portal vein or lymphatic cause the intrahepatic vein thrombosis and its surrounding inflammation, liver parenchymal necrosis, forming intrahepatic abscess right lobe. And can emboli forms of inflow of the lung, brain, such as the formation of migratory abscess. The the intestinal trophozoites also directly spread and surrounding tissue formation rectovaginal fistula, or a variety of lesions of the skin and mucosal ulcers. Individual cases can cause intestinal bleeding, intestinal perforation with peritonitis, appendiceal inflammation.
Visible under a microscope necrosis its primary lesions, lymphocytes, and a small amount of neutrophil infiltration. Serious bacterial infection was acute diffuse inflammatory changes, more inflammatory cell infiltration and edema, necrosis change. The lesion site visible multiple amoebic trophozoites, mostly gathered at the edges of the ulcer.
Amoebic dysentery - pathology classificationCommon type: onset and more slowly. Often abdominal pain, diarrhea. Diarrhea several times a day to more than ten times. The tenesmus varying degrees. The defecate medium, often pus or mucus typical stool was jam-like, corrupt stench. Can also be expressed as a simple diarrhea. Right lower quadrant tenderness. The course of a few days or a few weeks to relieve itself, if not treated, easy to relapse.
Fulminant: rare. Rapid onset, high fever, chills, diarrhea Day festivals worldwide, will be before a severe abdominal cramps, tenesmus obvious. The stool mucus bloody or blood samples, Stink. And vomiting, dehydration, rapid collapse. The physical examination see significant abdominal distension, diffuse abdominal tenderness, hepatomegaly. Not timely rescue, the concurrent intestinal bleeding, intestinal perforation, can cause death.
Chronic: symptoms persist or recurrent. Often abdominal pain, bloating, alternating diarrhea and constipation. Due to long-term bowel dysfunction in patients with weight loss, anemia, malnutrition or symptoms of neurasthenia. Colon wall thickening even palpable block of material, and tenderness.
Amoebic dysentery - Clinical performanceAmoebic dysentery average incubation period of 1 to 2 weeks (4 days to a few months), there are different types of clinical manifestations.(A) asymptomatic (cysts carriers) This type of clinical often not symptoms, the amoeba cysts found multiple Feces.
(B) The general type of onset and more slowly, systemic poisoning mild symptoms, often without fever, mild abdominal pain, diarrhea, a day will be about 10 times more than in moderate amount, blood and mucus, blood and necrotic tissue was jam mixed kind, with the stench of corruption, containing the dysentery amoeba trophozoites with a large number of red blood cells piles, one of its features. Low lesion with tenesmus sense. Abdominal tenderness in the right side of the main. Above symptoms and relieve itself. Can also be due to inadequate treatment of relapse.
(C) strong light seen in physical symptoms are mild, daily schedules thin paste or dilute water will be within 3 to 5 times, or diarrhea alternating with constipation appears, no diarrhea, only a sense of abdominal discomfort or pain, stool occasionally mucus or a small amount of blood to be investigated and the disease cysts and trophozoites. Without complications, the prognosis is good.
(D) fulminant rare, severe infection of the pathogen, or concurrent intestinal bacterial infections, and physical weakness, was fulminant. Abrupt onset of obvious symptoms of poisoning, chills, high fever, delirium, toxic intestinal paralysis. Severe abdominal pain and tenesmus, frequent diarrhea, dozens of times a day, or even incontinence, fecal, bloody, wash the flesh water or dilute watery, resembling acute dysentery, but fecal Stink, amebic trophozoites containing a large number of activities for its unique. Abdominal tenderness evident. To peripheral circulatory disorders often due to dehydration, or accompanied by a disturbance of consciousness, and even intestinal bleeding, intestinal perforation, peritonitis and other complications, the prognosis is poor.
(E) chronic type often due to improper treatment of the acute phase alternating diarrhea and constipation occur, so that the clinical symptoms of recurrent, persistent or February unhealed. Often cold, tired, eating carelessly and attack. Patients often feel lower abdominal pain, the long of fatigue, anemia and malnutrition. Right lower quadrant and thickening of the colon, mild tenderness; liver enlargement accompanied by tenderness. Feces mixed with pus and blood, trophozoites, sometimes with cysts.
(F) other type amebiasis visible urinary tract, reproductive system, skin infections, etc., but rare. Also the onset of complications, easily misdiagnosed.
Amoebic dysentery - etiology and pathogenesisEntamoeba histolytica life history, including trophozoites and cysts Phase. The basic process of the life history: cysts → trophozoites → cysts. Trophozoites have large, the small trophozoites stars, small trophozoites (luminal) of a diameter of about 10 ~~ 20μm, there is a single vesicular nucleus; large trophozoites (tissue-type) having a diameter of 20-40μm, cytosolic ectoplasm transparent, endoplasmic thick, which can contain glycogen, phagocytosis of red blood cells and tissue debris. Trophozoites of amoeba pathogenic stage, but non-infectious. Cysts seen of chronic amoebic ill or cysts carriers forming stool, a diameter of 5-20μm, Mature cysts 4 nuclear. The cyst is the infectious stage of the parasite.
Intestinal amoebiasis Entamoeba histolytica invasion of the colon wall diseases caused by oral infection, the main symptoms of diarrhea, abdominal pain, and it is also known as amoebic dysentery. 4 nuclear cysts with fecal contaminated water or food into the digestive tract, and can withstand stomach acid digestion, passed the upper section of the stomach and small intestine, excystation alkaline digestive juice to the lower segment of the small intestine develop into 4 small nourish body. The small trophozoites second division under suitable conditions proliferation, and down to the colon with feces. When the body's defenses are weakened bowel dysfunction, small trophozoites into the intestinal mucosa, phagocytosis of red blood cells and tissue cells into trophozoites, and a large number of proliferation, destruction of intestinal tissue, and the formation of ulcers.
Entamoeba histolytica pathogenic mechanism is not yet fully understood. Trophozoite surface film adhesion protein play an important part in the process of parasite invasion of host intestinal wall. As its a glycoprotein called adhesins or lectins, receptor like binding occurs with the mammalian target cells glycocalyx of galactose or acetyl galactosamine (Gal / GalNAc), so that nourish The body is adhered to the target cell. The trophozoite also secrete a into holes peptide and a variety of proteolytic enzymes. The former can be embedded in the target cell membrane and the target cell membrane permeability changes, dissolution occurs so that the cell host tissue; the latter can be dissolved. These factors are conducive to Entamoeba histolytica trophozoites invade host organization, the portal of entry is the opening of the midgut gland. From all aspects of the host pathogenic factors may include changes in innate susceptible hormones (such as adrenaline), the intestine environment (pH, redox potential, intestinal flora, etc.), malnutrition, primary and successors onset immunodeficiency and comorbidity. [1]
Amoebic dysentery - concurrent symptomsAmoebic dysentery complications points enteral, parenteral two categories:(A) of the intestinal mucosa ulceration depth of myometrial invasion and vascular complications when, can cause varying degrees of intestinal bleeding and intestinal perforation, the acute perforation may occur diffuse peritonitis or abdominal abscess. Chronic perforation is more common than acute perforation. Abdominal X-ray examination see subdiaphragmatic free gas can be diagnosed. Can also cause appendicitis. Amoeba tumors (colon granuloma), but not chronic inflammatory reactions, and the formation of granulomas, colon wall, can cause intussusception or obstruction. Biopsy help diagnosis.
(B) parenteral complications are the most common liver abscess, abscess perforation can be extended to nearby tissues and organs. By the trail directly involving the brain, lung, testicular, prostate, ovarian and other.
Amebic liver abscess (Amoebicliverabscess) can occur in the whole process of the disease, or the disease after a few weeks to several years. More to the long-term irregular fever onset, body temperature up to 39 ℃, common remittent fever, often accompanied by the right upper quadrant or right lower chest pain, liver enlargement of significant main clinical manifestations of tenderness. Abscess mostly single, mostly in the right lobe of the liver, because with the right lobe of the 4/5 of the entire liver volume, and intestinal lesions in the ileocecal department most of the blood circulation through the superior mesenteric vein inflow of the right lobe of the liver. Significant local symptoms and signs of liver abscess in the left lobe, in a relatively short time, but the diagnosis more difficult. Abscess superficial can have localized tenderness or with a sense of volatility, see this time line transhepatic liver-colored, fishy smell of pus containing dissolved necrosis of liver cells, red blood cells, fat, Charcot-Leyden crystals, trophozoites not more common, can be found in the abscess cavity wall, but did not find cysts. If the bacterial infection, abscess, yellow-green or yellow-white pus.
Chronic cases of fever and more obvious, weight loss, anemia, malnutrition, edema. Peripheral blood: WBC count increased the early period, the latter can be reduced to normal. The stool examination protozoa positive rate is not high. C duodenal drainage tube bile visible trophozoites.
Liver function tests are mostly normal transaminase, serum cholinesterase decreased slightly elevated alkaline phosphatase. X-ray examination showed the right diaphragm elevation, activity limitation, The local uplift diagnostic significance. Left lobe abscess, barium meal examination showed the pressure on the lesser curvature of the stomach and gastric body to the left phenomenon. B-type ultrasonic isotope liver scan, CT scan, magnetic resonance imaging and other tests to help diagnose.
Amoebic lung abscess and more swollen abscess secondary to its main symptoms of bacterial lung abscess, bronchiectasis similar. , And coughed up a lot of brown pus concurrent bronchopulmonary fistula. Can be complicated by pleurisy with pleural effusion, as was brown in the diagnosis. Amebic pericarditis is rare and may be left lobe amoebic liver abscess penetration pericardial Erzhi. Symptoms similar to bacterial pericarditis, is the most dangerous complications of the disease.
Amoebic dysentery intestinal complications and extraintestinal complications.
(1) intestinal complications
The ① intestinal bleeding: intestinal wall ulcer involving the blood vessels, can cause intestinal bleeding. How much blood loss ranged bleeding patients often appear pale, pulse breakdown and drop in blood pressure, hemorrhagic shock performance.② intestinal perforation: more common in fulminant. Perforation site cecum, appendix, and ascending colon is more common. Acute perforated diffuse peritonitis can cause serious illness. Chronic perforation caused by the surrounding tissue adhesion, the formation of a local abscess.③ appendicitis: of amebic appendicitis symptoms similar to ordinary appendicitis, easy to abscess formation, if chronic diarrhea or amoebic dysentery history, found in the feces of the amoeba trophozoites or cysts, contribute to the differential diagnosis of the two.The ④ nondysenteric colonic lesions: caused by proliferative lesions, including ameboma, intestinal of amebic granuloma and fibrous stenosis. The inflammatory pseudotumor ameboma the large intestine, abdominal pain and bowel habits change the most part with intermittent dysentery, can induce intussusception and intestinal obstruction, the main signs: the right iliac palpable removable, smooth goose egg-shaped or bowels like blocks, X-line to see occupying lesions have a good effect against amoebic treatment.
(2) parenteral complications: amoeba trophozoites from the intestinal blood  lymphatic spread to distant organs and cause a variety of parenteral complications, such as the liver, lungs, pleura, pericardium, brain, peritoneum and genitourinary Road to form abscesses or ulcers, which is the most common liver abscess.
Amoebic dysentery - Differential DiagnosisThe diagnosis of amoebic dysentery (a)1, the clinical manifestations of slow onset, mild symptoms, diarrhea, fewer and dark red Secheng sauce-like droppings of the disease should be considered.
2 detection of Entamoeba histolytica, fecal examination under a microscope for diagnosis important basis. The bloody mucus loose stools Yi find trophozoites, cysts fecal matter part is easy to find.
3, sigmoidoscopy varying sizes scattered in the shape of the latent ulcer edge slightly elevated, blush, ulcers mostly normal mucosa. Scraped from the ulcer specimens microscopic examination found the pathogen more opportunities.
X-ray barium enema intestinal stenosis, ameboma a certain value.
The serological examination available amoebic pure antigen detection of specific antibodies, formation of antibodies vivo invasive lesions square cyst carriers antibody test was negative. Commonly indirect hemagglutination, ELISA, indirect fluorescent antibody, counter immuno electrophoresis, agar diffusion precipitation test.
(B) Differential Diagnosis
This disease and bacillary dysentery. Japan schistosomiasis, Gram giardiasis, intestinal tuberculosis, nonspecific ulcerative colitis differentiated. Amebic liver abscess subphrenic abscess, cholelithiasis identification of primary liver cancer, liver bursitis.
Amoebic dysentery - treatment methodGeneral treatment of amoebic dysentery (a)
Acute phase should rest in bed, gut isolation until the symptoms disappear, stool for three consecutive times finding out trophozoites and cysts, better nutrition, infusion or transfusion when necessary. With acute and chronic dysentery.
(B) the pathogen treatment
Metronidazole (Flagyl) Adult 0.4 ~ 0.6g, 3 times a day, and even served 5 to 7 days, children 15mg/kg, 3 times a day, once every 3 to 5 days. Not oral administration the available intravenous injection of 0.5%, can change oral oral. Medication during Jijiu, pregnant women and lactating women Jifu. Chronic cases, can be used to retain the enema. Attention to the side effects of the drug: nausea, dizziness, palpitations, leukopenia, and other.
2, the tinidazole (metronidazole sulfonyl imidazole) adult daily 2.0g daily for children 50mg/kg every morning serving Dayton, the 3rd one course of treatment. Thiazole can also use the piperazine denitrification 0.18,3 / d, children 10mg / (kg · d), treatment 7 ~~ 10d, when necessary, and even served 1 to 2 courses.
3, dehydroepiandrosterone emetine (dehydroepiandrosterone ipecac base): Adults 60 ~ 80mg, pediatric lmg / kg, twice a day, intramuscular injection, once every 5 to 10 days. More toxic hydrochloric emetine small, less serious side effects.
4 emetine (hydrochloric acid emetine): the adult 60mg, pediatric lmg / kg, twice a day, intramuscular injection on the 6th. To the onset of myocardial damage and toxic myositis, treatment should be bed rest, often observe blood pressure, pulse, electrocardiogram, prior to each injection to listen to heart sounds, blood pressure, and are subject to change, you should consider the reduction or withdrawal. Pregnant women and heart, kidney patients hanged. Large trophozoites have a direct role in the killing, and quickly to control the symptoms of acute dysentery and extraintestinal complications, but the small intestinal lumen trophozoites and cysts invalid.
5 dichloro San: 0.5g, 3 times a day, and even served on the 10th.

In the application of these drugs a course, you are required to use one of the following drugs continue treatment. Metronidazole can be used alone.
① sulfonamide iodine quinoline: 0.5 g / d, used in conjunction 10d.② Iodoquinol: 0.6g, 3 / d, and even served 20d.③ paromomycin: 0.5g / d, 10d for a treatment course. Fulminant optional By Miding 3d, a course of metronidazole, while adding norfloxacin or tobramycin or ceftriaxone latamoxef to combat secondary bacterial infection.
Chinese medicine treatment: alone or in combination with other anti-amoebic drugs.
① Differential Treatment: the same bacillary dysentery.
(2) selection of effective herbs such as Pulsatilla, javanica, garlic, etc. in the light of local conditions.
7, with cysts treatment: can I dichloro scattered 0.5g 3 / d, a total of 10d; or metronidazole 0.4g / d for 5 days.
8, contact medical observation necessary clothing metronidazole a course of treatment. Stool changes, should be promptly smear or culture dysentery amoeba.

(C) the treatment of amoebic liver abscess
1, metronidazole 0.4g, 3 times a day, the inflammation of the course of 2 to 3 weeks, a abscess period of 4 weeks, abscess smaller puncture pus.
2, phosphate chloroquine 0.5g, 3 times a day, after 2 0.25g daily three times, once every three weeks. The medication is not effective seven days can change his drug.
3, liver puncture pus while medication can puncture the abscess, the abscess is greater pus into the Tugen base 30 to 60mg. The pus should be culture sensitive antibiotics should be added, if secondary bacterial infection.
Amoebic dysentery - herbal unilateralAmoebic dysentery Chinese herbal treatment of amoebic dysentery javanica, Pulsatilla and garlic.(1) Brucea javanica: evergreen shrub or small tree of Simaroubaceae Brucea javanica mature seeds, mainly produced in Guangxi, Guangdong and other provinces. Autumn fruit ripening harvest, dried, shelled Ren the dry longan meat and encapsulated swallow, or pressure to oil, made of pills or tablets clothing. Taste the bitter cold of the character, in the final of the large intestine, the liver. Detoxify, anti-malaria cut malaria, the effect of corrosion wart. Oral available to treat Noxious Heat dysentery, cold product Jiu Li and dysentery, topical can cure corns wart. According to previous treatment experience cold product Jiu Li, Modern clinical for the treatment of amoebic dysentery, to take oral enema and methods. Oral, each adult 10 to 20, children 2 years old, swallow capsules, 3 times daily for 7 to 10 days for a course of treatment, and 20 Brucea javanica pestle, adding 1% sodium bicarbonate solution 200ml, soak 2 hours as a retention enema daily, has a good effect on the acute and chronic amoebic dysentery. Its mechanism of action is of Brucea javanica its active ingredients the amoeba have to kill and inhibition. The goods on the gastrointestinal tract, liver and kidney damage are not appropriate to use Jiufu. Gastrointestinal bleeding and liver kidney patients, should be hanged or caution.
(2) Pulsatilla: Ranunculaceae perennial herb Pulsatilla root. Distributed in Northeast China, Inner Mongolia, and North. Spring before flowering or late fall when the leaves yellow before harvest to retain the root head white fluff, remove the leaves and residual stems and fibrous roots, wash dirt, dried, raw with. The medicinal taste the bitter cold, owned by the large intestine through, there are detoxification, cooling blood Zhilishen efficacy, clinical commonly used to treat hot Noxious dysentery, Qin skin compatibility Jiantang wash available Yin Yang, trichomonas vaginitis. FDA treatment of bacillary dysentery and amoebic dysentery, and have good results. Such as the treatment of amoebic dysentery, Single Pulsatilla 15 ~ 30g, decoction, 3 service points, seven days for a course of treatment; seriously ill by another FDA 30 ~ 60g, Jianshui retention enema daily, quite good effect. The main mechanism of Pulsatilla treatment of amoebic dysentery: Pulsatilla decoction and saponins contained obvious anti-amoeba role.
(3) Garlic: Liliaceae perennial herb of fresh garlic stems. All over the country set out to training. May leaf blight excavation, dry medicine. The potency Weixin temperature, spleen, stomach, lung meridian. The scattered carbuncle swelling, detoxification insecticidal efficacy, clinical commonly used to treat a carbuncle swollen sore ringworm sores pruritus, cough tuberculosis Dayton, dysentery, diarrhea, hookworm and enterobiasis of. Garlic on acute and chronic amoebic dysentery are effective in the prevention and control of dual-purpose. The single fresh garlic, or 10 ~ 15g garlic mash, sugar water, blunt. Garlic syrup, or oral administration of 10% per serving 5 ~ 20ml, 3 times day. Or garlic extract 100ml of 10% retention enema once a day, once every 6 days.
Amoebic dysentery - formation characteristicsAmoebic dysentery, bacillary dysentery in the early stages of disease usually presents with diarrhea without blood and pus, later typical pus and blood will be the cause of this phenomenon is caused by the intestinal pathological changes of bacillary dysentery patients.Shigella through the mouth into the digestive tract, in the stomach by gastric acid that kill. People lowered immunity, Shigella stomach into the small intestine, the large population of some dead bacteria release endotoxin, stimulate the intestinal wall so that the increased permeability. Toxins being absorbed into the bloodstream, which may eventually be discharged from the colon mucosa, irritable colon and cause mucosal damage, on this basis, Shigella, and intestinal bacteria in the mucosal surface and submucosal breeding further disruptive, causing inflammation reaction.
Early onset of secretion of Shigella endotoxin and inflammation stimulate the intestinal wall nerve endings, causing intestinal cramps, bowel movements increased intestinal wall to absorb moisture and lower bowel wall vascular serous exudate, and suffered from diarrhea; Thereafter, intestinal diffuse congestion and edema, massive neutrophil infiltration, accompanied by a lot of mucus and fibrin exudate, and finally to the formation of ulcers, bleeding, mucous pus before.
Amoebic dysentery stool characteristics with intestinal lesions into different, some form of liquid, some were semi-mobile, and even shaped stool, which can be no blood or mucus attached. Dysentery attack, the amoeba in intramural breeding, causing tissue necrosis, or invasion deeper, more bleeding, necrotic tissue mixed with blood with stool, constitutes a amoebic dysentery unique jam color, kind of frozen fish, the stench of fecal matter more bloody stool.
Amoebic dysentery - prevention and control points1 pay attention to food hygiene, personal hygiene and civilized way of life, do not drink unboiled water, do not eat unclean fruits and raw vegetables, and develop hors d'health habits food wash their hands before and after production.2. Strengthen manure management, health management of livestock pens, according to local conditions good manure treatment, improved sanitation.Protection of public water to prevent fecal contamination. Drinking water should be boiled.Strengthen the health management of the canteen. Operation of food production and staff should have health supervision measures.5. Vigorously fight flies, cockroaches, fly-proof cover or other measures to avoid contamination of the food is.6. Patients should be treated quickly, outbreaks have been reported in infectious disease management approach, disinfection, isolation processing. Should be made to check on family members or contacts.The number of cases in an area, to quickly for laboratory tests to confirm the diagnosis and for epidemiological investigation and take appropriate measures.
Amoebic dysentery - Safety Tips1. Of the disease throughout the world, prevalent in tropical and subtropical regions. China's more common in the north. The incidence rate in rural areas than in urban; men than in women, adults more often than children, most of them are sporadic, summer and autumn higher incidence of water pollution and other factors, even the outbreak.
2. Chronic amoebic dysentery usually caused by the acute phase of improper treatment or patients with poor physical symptoms recurring more than 2 months delayed healing, exacerbation of clinical symptoms similar to patients with acute and chronic illness anemia, weight loss, nutrition bad and hepatomegaly.
3. Modern medicine of the disease of chronic disease and asymptomatic parasite is the main source of infection of the disease, its discharge feces amoeba cysts by hands, food, drinking water, flies and cockroaches pathway by Mouth digestive tract into the body.